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Superficial fungal infection of the skin, commonly called ringworm or dermatophytosis, has become one of the biggest dermatology problems in India over the last decade. What used to be a simple condition cured in two weeks is now frequently extensive, intensely itchy, recurrent and resistant to standard treatment. The reason is well established and worth stating plainly: widespread over-the-counter use of combination creams containing potent steroids.
Combination creams sold across Indian pharmacies typically contain a potent steroid mixed with an antifungal and often an antibiotic. The steroid suppresses redness and itching within a day, so patients believe it is working. But the steroid also suppresses the local immune response that keeps the fungus in check, allowing it to spread widely under the surface with the classic ring border erased. This is called tinea incognito, or steroid-modified tinea, and it now accounts for a large share of cases. Repeated use has also contributed to genuinely resistant strains such as Trichophyton indotineae.
If you are using a combination cream, stop it and see a dermatologist. These preparations are the main driver of the recurrence patients experience.
1. Tinea corporis: Body ringworm, with expanding rings and central clearing.
2. Tinea cruris: Groin and inner thigh, extremely common in hot humid weather.
3. Tinea pedis: Athlete’s foot, between the toes.
4. Tinea capitis: Scalp, mostly in children, causing patchy hair loss and needing oral treatment.
5. Tinea unguium (onychomycosis): Nails, thickened, discoloured and crumbly, requiring months of oral therapy.
6. Candidiasis: In skin folds, under the breasts, in the groin and armpits, particularly in diabetics.
7. Tinea versicolor: Hypopigmented or brown scaly patches on the chest, back and shoulders.
1. Heat, humidity and sweating, which is why incidence peaks in the Indian summer and monsoon
2. Tight synthetic clothing and prolonged wearing of damp clothes
3. Sharing towels, clothing, combs and footwear, and household spread among family members
4. Uncontrolled diabetes and obesity, especially in skin folds
5. Immunosuppression from disease or medication
6. Prior use of steroid or combination creams
7. Public gyms, swimming pools and shared bathrooms
See a dermatologist if the rash is spreading, has recurred after previous treatment, involves large areas, the scalp or the nails, or if you have been using a combination cream. Diabetics with fungal infection should be treated promptly. Do not continue self-medicating, as each cycle of steroid use makes the next episode harder to clear.
1. Stop all steroid and combination creams immediately. This is the single most important step and symptoms may transiently worsen before improving.
2. Confirm the diagnosis: KOH microscopy, and fungal culture in resistant or atypical cases.
3. Topical antifungals: Plain terbinafine, luliconazole, amorolfine or ketoconazole, applied to the lesion and 2 cm of surrounding normal-looking skin.
4. Oral antifungals: Itraconazole or terbinafine, for an adequate duration. Under-treatment is a major cause of recurrence. Extensive or resistant disease often needs 4 to 8 weeks or longer, not the one week patients typically take.
5. Complete the full course: Treatment continues for at least two weeks after the rash appears to have cleared, since fungal elements persist after visible clearing.
6. Treat the whole household simultaneously: Untreated family members reinfect the treated patient, which is a very common reason for apparent treatment failure.
7. Environmental measures: Hot washing and sun drying of clothes and bed linen, loose cotton clothing, keeping skin folds dry, antifungal dusting powder and not sharing towels.
8. Control blood sugar and treat any co-existing skin condition.
Untreated or steroid-treated ringworm spreads relentlessly, and patients who began with a single coin-sized patch commonly present months later with involvement of the entire groin, buttocks, trunk and thighs. Chronic scratching causes lichenification and post-inflammatory pigmentation that persists long after the fungus is cleared. Steroid-damaged skin develops striae, thinning and visible vessels that are permanent. And every incomplete course selects for more resistant organisms, which is precisely how India arrived at the current situation. Correct, adequately long treatment on the first attempt is the whole solution.

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